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Although reaching a weight of pounds may have been caused by consuming, say, calories more than were used each day, losing that weight requires much larger reductions in calorie intake. According to Dr Hall's calculations, an extra calories a day are now maintaining the new higher weight. Dr Hall noted that typical weight-loss programs result in significant losses over a period of six to eight months, followed by gradual weight regain in the years that follow.
Although consuming an extra calories a day would not show up right away as weight gain, it does over time. And it happens more slowly for the obese person than for someone who is lean, Dr Hall said, because the obese person's body requires more calories to maintain the extra pounds.
Page : 1 2 3 Single Page Format. They do not represent the views or opinions of The Indian Express Group or its staff. But the hormonal changes could just as well have followed from the weight loss. Leptin levels in the plasma are known to drop during a very low-calorie diet, as well as when a person has been shedding fat.
Contestants on The Biggest Loser , for example, saw their concentrations founder by almost 95 percent over the course of the weight-loss competition. That changes such as these might still be detectable, to some degree, 12 months down the road could just as well reflect the fact that the patients had maintained some degree of weight loss across that time, too. A more comprehensive theory of weight regain, accounting for a broad array of mechanisms, may help address some of the confusion in this field.
Researchers Marleen van Baak and Edwin Mariman of Maastricht University, for example, have proposed that the compensatory reflex begins with changes to the shape of fat cells. As these cells drain and shrink, their membranes pull away against the points of adhesion to the nearby extracellular matrix, creating mechanical stress.
According to their preliminary model, which is based on both in vitro studies of adipocytes and examinations of protein expression during and after weight loss, the mechanical tension that shedding weight creates at the fat-cell membranes inhibits further fat release and primes those cells to be filled again. At the same time, they theorize caloric restriction may deprive adipose tissue of the energy it would need to relieve this stress through remodeling of the extracellular matrix.
This was followed by a brief weight-stabilization period in which they received about as many calories as they would need to keep a constant weight and then further check-ins for the next nine months. The team took biopsies of adipose tissue at the end of each study phase, measured changes to its gene activity, and checked to see which, if any, might be correlated with weight regain.
In a subgroup analysis of the crash diet participants, 15 genes related to the extracellular matrix were identified, and 8 more associated with stress response. Others are looking for answers in the genome. But a genome-wide association study to determine whether genes that have been linked to the development of obesity might also be predictive of weight regain failed to turn up any positive results. That could be on account of its insufficient sample size, McCaffery explains.
The study had about 3, people in the weight-loss condition, whereas similar studies of the genetics of obesity have been far larger in scope. The one point on which nearly all researchers agree is that the physiology of weight regain, like the physiology of obesity itself, is almost certain to reflect a very complicated mix of factors ranging from genetics to behavior and the environment.
Indeed, some degree of rebound may be more or less inevitable for the majority of dieters. But even that news may not be as bad as it seems. Just last year, a team of researchers at the University of Alabama at Birmingham, led by David Allison, put out a rodent study of a provocative idea: what if there were lasting benefits to losing weight—even when that weight is almost certain to be regained?
At the end of the study, the mice that remained obese throughout the experiment had markedly increased mortality: they lived, on average, for just 21 months, as compared to the month average lifespan of the mice that had been put on the most extreme diets and kept at a normal weight. More surprising was the fact that the yo-yo mice also gained longevity, by virtue of their weight cycling: they lived an average of 23 months, about the same as the mice that were kept under chronic, moderate calorie restriction.
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